Therefore, it can be conceivable the JIP1/3 → ASK1 → MKK4 signaling module may serve as a conduit to activate JNK when endosomal trafficking is disrupted and glucose amounts fall in the course of the early levels of methuosis. Our finding that MKK4 was robustly activated through the methuosis-inducing IPPs, https://salmeterolxinafoate55442.blogolize.com/helping-the-others-realize-the-advantages-of-triptolide-65510303